Upregulation of PI3K/AKT/mTOR, FABP5 and PPARβ/δ in human psoriasis and imiquimodinduced murine psoriasiform dermatitis model

Christopher-Chamcheu, Jean; Siddiqui, Imtiaz; Mukhtar, Hasan; Chaves-Rodríguez, María Inés

Upregulation of PI3K/AKT/mTOR, FABP5 and PPARβ/δ in human psoriasis and imiquimodinduced murine psoriasiform dermatitis model

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upregulation-human-psoriasis.pdf (221.41 KB)

Date

2016

Authors

Christopher-Chamcheu, Jean

Siddiqui, Imtiaz

Mukhtar, Hasan

Chaves-Rodríguez, María Inés

Publisher

Acta Dermato-Venereologica

Abstract

Psoriasis is a common, and currently incurable chronic immune-mediated skin disease, with incompletely understood etiology partially due to the unavailability of animal models that can emulate major features of the disease.

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Artículo científico

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Farmacología, Animales, Enfermedades, Ratones, Metabolismo, Research Subject Categories::MEDICINE::Dermatology and venerology,clinical genetics, internal medicine::Dermatology and venerology

Citation

Christopher-Chamcheu, J., Chaves, M., Siddiqui, I., & Mukhtar, H. (2016). Upregulation of PI3K/AKT/mTOR, FABP5 and PPARβ/δ in human psoriasis and imiquimodinduced murine psoriasiform dermatitis model. Acta Dermato-Venereologica, 96(6).

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https://hdl.handle.net/2238/9933

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Upregulation of PI3K/AKT/mTOR, FABP5 and PPARβ/δ in human psoriasis and imiquimodinduced murine psoriasiform dermatitis model

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